At normal levels, ADAR editing contributes to transcriptome diversity and brain health, but ADAR editing can become dysregulated leading to neurological and psychological disorders. ADAR editing is observed in both mouse and human. This project aims to identify orthologous genes across mouse and human genomes using Ensembl and MGI to identify specific codons that correspond to human editing targets. Additionally, a literature review is done to evaluate the current state of knowledge about edited genes and the potential link to functions related to neurological and psychological health. The project focuses on 90+ human excitome genes that play a role in the excitatory pathways of the nervous system, e.g., GRIA2. Collected information can be utilized to better understand the evolution of genes with important functions in mammalian brains.